I. Train to Your Nature
II. The Ten Physical Capacities
III. Measuring Your Baseline
IV. Training Endurance
V. Training Strength and Power
VI. Gymnastics and Bodyweight Progression
VII. Kettlebell Training
VIII. Training Mobility and Movement Quality
IX. Recovery and Adaptation
X. Training Fasted
XI. The Gist
Before we get into the methodology, let’s orient this page…
We all have a greater genetic disposition for certain exercises over others. Some bodies are built for explosive power. Some are built for endurance. Some recover quickly from heavy loads. Some have superior balance and proprioception from the moment they pick up a sport. While it’s possible to discover what types of exercise you’d be better suited for by getting a genetic screen or muscle biopsy, it’s just as easy to go by what exercises you “like.”
More often than not, the exercises you like tend to be the ones you’ve shown promise in or have proven to be good at naturally (social reinforcement is a different story). The positive reinforcement from performing a movement exceptionally well is typically enough of a reward to keep doing it consistently. Pay attention to how quickly your muscles activate, how quickly you develop strength, and how quickly your cardiovascular fitness improves compared with your peers to gauge what you may be better suited for.
It’s possible to alter the composition of your muscle fibres to become more suited to an exercise you weren’t always good at, but if longevity is the goal, it makes more sense to attempt movements your body genetically anticipates you to attempt. We all love a success story about somebody who achieved greatness despite terrible odds, but this is the exception rather than the rule. The entire body works systematically and requires a huge energy demand in order to change its predisposition. Our genetic predisposition is due to generations of environmental adaptation and evolutionary selection. Our bodies are programmed to expect certain physical stimuli and environmental cues, which atrophy without exposure (a common circumstance given our sedentary modern-day lifestyles).
Work smart, not hard, and try not to do something just because somebody told you not to. Then again, breaking the boundaries of what we know is what leads to progress. So, if your goal is longevity, do what you’re good at. If your goal is exploration and to challenge yourself mentally, do whatever you want. Just try to understand that you will elicit greater personal authenticity and psychological alignment by understanding who you are and what you are capable of.
The pages that follow assume you’ve done some honest self-assessment on this front. Don’t let someone else’s protocol dictate your training when your goals and genetic predispositions are different from theirs.
Physical performance isn’t a single thing. It’s a collection of distinct capacities, each trainable, each with its own physiology, each requiring specific stimulus to improve. The widely adopted framework (originally formalised by Greg Glassman of CrossFit, drawing on Jim Cawley’s earlier work, with substantial overlap with the National Strength and Conditioning Association taxonomy) breaks human physical capacity into ten components:
A well-rounded human is reasonably competent across all ten. A specialist athlete will be exceptional in two or three areas and weaker in the rest. Most modern adults are weak in all ten, having gradually lost capacity through a sedentary lifestyle. The training framework that follows addresses each capacity, but the practical recommendation for most adults is to address the worst three first rather than chasing marginal gains in the strongest two.
You can’t intentionally train what you don’t measure. The following tests, drawn from the sports science and clinical exercise physiology literature, give you a baseline across most of the ten capacities. Pick the ones relevant to your goals. Re-test every 8-12 weeks to confirm whether your training is producing the adaptations you intended.
The gold standard is a laboratory VO2 max test on a treadmill or cycle ergometer with gas exchange analysis. For most people, two field tests do nearly as well:
UKK 2km walk test for estimated VO2 max: Walk 2 km on a flat surface as fast as possible. Record time and heart rate at the finish. Adequate accuracy is achieved when the finishing heart rate is at least 80% of maximum. Not recommended for people with very high fitness (the test maxes out).
Cooper test (12-minute run): Run as far as possible in 12 minutes on a track or flat surface. The result correlates well with maximal oxygen uptake for trained runners. Better suited to people who actually run as part of their training; less reliable for people whose primary endurance modality is cycling, swimming, or rowing.
Clinical exercise stress test (exercise ECG): Usually performed on a cycle ergometer in a clinical setting. Detects cardiovascular disease potential while measuring aerobic fitness and anaerobic force generation. Arterial blood oxygen and lung function may also be measured. The more comprehensive version (running spiroergometry) measures oxygen consumption, CO2 production, anaerobic threshold, and (with arterial blood draw) lactate response.
Wingate test: A cycle ergometer test of anaerobic capacity. Five to ten minutes of low-power warm-up followed by 30 seconds of pedalling at maximal power against a standardised load. Best performed in the afternoon or evening during peak power times.
Measured outputs:
MART (maximal anaerobic running test): Tests properties related to endurance and speed in running athletes.
RAST (running-based anaerobic sprint test): Similar to the Wingate but for ball sports. Six 35-metre maximal sprints. Power calculated as: bodyweight × distance² / time³
The Functional Movement Screen (FMS), developed by Gray Cook and Lee Burton, has become the most widely used clinical mobility assessment. Seven movements scored 0-3 each, with a total maximum of 21:
Scores below 14 are associated with substantially increased injury risk in athletic populations. The FMS isn’t a measure of fitness; it’s a screen for movement quality and asymmetries.
Simpler home mobility tests for specific joints:
Grip strength has been identified across multiple large cohort studies (most notably the PURE study, Leong et al. The Lancet 2015) as one of the strongest non-invasive predictors of all-cause mortality, cardiovascular events, and disability in older age. Stronger than systolic blood pressure in some analyses. Translates well into deadlift and pull-up strength as a secondary measure, which is pretty obvious why if you think about it.
Surface EMG (electromyography) can measure:
This is laboratory-grade testing that most people will never need. Useful for athletes working with sports scientists on biomechanical issues.
Objective tools:
Subjective tools:
Factors affecting recovery:
The HRV framework, in particular, has been a substantial methodological advance. Cleared a substantial portion of guesswork out of training load management for athletes; emerging consumer wearables (Whoop, Oura, Garmin, Polar) have brought it within reach for the rest of us. We’ll cover the specifics later on this page.
Definition: The maximum amount of oxygen you can utilise, an amount called VO2 max (also called maximal oxygen consumption or peak oxygen uptake). Performance depends on the respiratory and cardiovascular systems and on energy management in the muscles (their ability to convert fat and carbohydrate into energy). This is determined by the number of mitochondria, the number of capillaries in the muscles, and various metabolic pathways (glycolysis, Krebs cycle, oxidative phosphorylation).
The minimum effective dose for maintaining VO2 max appears to be five 4-minute high-intensity rounds at 87-97% of maximum heart rate, with approximately 4 minutes of rest or low activity between rounds. This is the “4×4” protocol popularised by the Norwegian endurance research community and Peter Attia.
The polarised training framework suggests roughly 80% of weekly endurance volume in zone 1-2 (easy, conversational pace, predominantly fat-burning) and 20% in zone 4-5 (high intensity, at or near VO2 max). Very little time in the moderate-intensity middle.
The standard public health recommendation is 2.5 hours per week of moderate-intensity activity. Walking, cycling, swimming, hiking, and even heavy house and yard work qualify. Running, cross-country skiing, fast cycling, or ball games push intensity to drive additional adaptations.
Endurance training can be broken down into:
The thresholds:
A practical zone framework synthesising the standard sports science taxonomy:
Zone 1/Basic Endurance 1 (Recovery):
Zone 2/Basic Endurance 2 (Aerobic Base):
Zone 3/Tempo Endurance 1:
Zone 4/Tempo Endurance 2:
Zone 5/Maximal Endurance:
Beyond Zone 5:
Common pitfalls: training at the same intensity over and over, training at the same pace, training too hard on lighter days, training too easy on hard days. Get variation.
Structural benefits of endurance training: increased heart volume and stroke volume, increased lung capacity, increased number of mitochondria, increased microvasculature (capillary density), lower resting blood pressure, lower resting heart rate, improved oxygen uptake, positive impact on anxiety and depression, balancing stress response, treatment and prevention of numerous chronic illnesses.
Disadvantages of excessive endurance exercise: cardiac remodelling and increased arrhythmia risk, repetitive strain injuries, impaired muscle mass and strength when chronic cardio crowds out resistance training.
85-95% of maximum heart rate in interval form. The rest phase is usually 60-70% of maximum heart rate. By varying the work phase from 10 seconds to 4 minutes, you can develop different energy systems. The biochemical effects on muscle cells (lactate, ATP, creatine phosphate, H+) don’t track tightly with rest interval length; the benefits of varying rest intervals come more from neurological, hormonal, and cardiovascular changes.
HIIT develops the cardiovascular and circulatory systems, maximal oxygen uptake, insulin sensitivity, sugar metabolism, and lactate tolerance. It increases mitochondria in muscle cells and the volume of oxidative enzymes. Fat oxidation benefits too.
Tabata protocol (Tabata et al. 1996, Medicine and Science in Sports and Exercise):
Gibala method (developed by Martin Gibala at McMaster):
Sprint Interval Training (SIT):
HIRT (High-Intensity Resistance Training): Shorter recovery periods are better at producing GH and improving muscular endurance.
Sample HIRT session:
Superset 1 (8-10 minutes without breaks):
Superset 2 (8-10 minutes without breaks):
Superset 3:
Physical strength is determined by two factors: the cross-sectional area of a muscle and the volume of muscle fibres plus their contractile intensity. Force generation hinges on the ability of the nervous system to command, recruit, and organise muscle fibres efficiently. The strength of connective tissue (tendons, fibrous tissues) also affects force production. Generation varies with cell type distribution, sex, age, hormonal balance, nervous system function, general health, and nutritional status.
Henneman’s size principle, formulated by Elwood Henneman in 1965, is the foundational principle for understanding how muscles produce force. Motor units are recruited in order from smallest (low threshold) to largest (high threshold) as force demand increases. This staircase pattern means you can’t deliberately recruit high-threshold motor units without sufficient force demand; high-threshold units only come online when low-threshold units are unable to meet the load. Heavy loads (above ~70% of 1RM) or near-failure efforts at lower loads are needed to recruit the high-threshold motor units that produce the largest hypertrophy and strength gains.
Key principles for strength training:
Loading and rep ranges:
Varying TUT duration affects different energy systems (ATP, creatine phosphate, anaerobic glycolysis). A set of slower repetitions of longer TUT performed to exhaustion is more effective for hypertrophy than faster reps for the same number of reps.
This is where the work of Brad Schoenfeld at CUNY Lehman College has substantially shifted the field over the past decade.
Henneman’s size principle in practice:
The three primary stimuli for muscle growth, as articulated by Schoenfeld’s earlier work:
Different rep ranges and tempos bias toward different stimuli. Maximum hypertrophy probably requires all three.
The minimum effective dose for maintaining muscle:
Roughly 5 sets per muscle group per week, trained near failure, appears sufficient to maintain muscle mass in trained individuals.
Isometric training:
May be used to promote recovery from injury. Can increase strength and muscle mass, but only at the joint angles trained. Dynamic muscular training is required for full range strength.
Eccentric quasi-isometric training:
Slow or almost static eccentric movement to strengthen muscles at all joint angles.
Super-slow repetitions:
Growth of satellite cells and muscle cell nuclei. Particularly useful for people over 50 (less injury risk than heavy, fast lifting). Drawback: weak development of maximal strength and lesser metabolic impact on energy expenditure and fat burning.
Super-slow eccentric repetitions (40X0 tempo):
Very slow eccentric followed by explosive concentric. Excellent for maximal muscle growth and tendon strengthening.
Negative repetitions:
Purely eccentric loading, allowing higher than 1RM weight to be used. Requires a spotter or safety bars. Very exhausting due to supramaximal loads. Use sparingly.
Pre-set bodyweight circuit (the “7-minute workout”):
Each exercise is 30 seconds with 10 seconds rest. The protocol from Klika and Jordan (2013, ACSM Health & Fitness Journal).
Gymnastics develops physical strength, coordination, balance, agility, muscular endurance, and flexibility simultaneously. Excellent foundation for children and remarkable rehabilitation for adults.
Easy:
Medium:
Difficult:
Strength, speed, balance, endurance, and maximal oxygen uptake.
Easy:
Medium:
Difficult:
Sample circuit (one exercise to the next with 30-60 second rest):
Warm-up: 5-10 minutes of slingshot, halo, light jogging, or burpees.
Working sets:
Optimal mobility is crucial for good posture and the prevention of incorrect positions during exercise. The training framework distinguishes between flexibility (passive range of motion) and mobility (active control through range of motion). For most adults, mobility is the most useful capacity to train.
Dynamic stretching programme (suitable as a warm-up before a workout):
The Huberman stretch protocol (drawn from his Neural Network Newsletter synthesis of the research literature):
Microstretching – research by Wyon et al. has shown that microstretching (30-40% of your maximum stretch intensity, where 100% is slightly painful) is more effective than aggressive stretching for long-term flexibility gains. Whatever stretching routine you adopt, you don’t need to push to a point of pain. Consistency and frequency are the variables that produce long-term flexibility gains, not intensity.
Static stretching protocol:
PNF (Proprioceptive Neuromuscular Facilitation), also called contract-relax stretching:
Antagonistic muscle group training:
Alternating exercises of antagonistic muscle groups (e.g., rows alternating with bench press) leverages reciprocal inhibition: contracting the biceps “relaxes” the triceps and vice versa. This allows higher-quality work in less time and reduces some forms of muscular tension.
Natural movement (Parkour-style training):
A useful adjunct to gym work for restoring movement patterns the body expects.
Deep bodyweight squat hold:
Hanging on a bar (passive):
Wall support hold:
Walking on all fours, jogging, sprints, and jumps:
Bodyweight exercises work muscle groups and certain functional muscle-tendon-fascia lines that conventional resistance training often misses. The Katy Bowman and Erwan Le Corre frameworks for natural movement (covered in Resources) provide more substantial protocols if this approach interests you.
The supercompensation theory:
Training consumes common resources, biochemical cascades, energy reserves, and the nervous system. Training represents a catabolic activity. The body needs rest, hydration, and nutrition to bounce back from the catabolic state. If recovery is optimal, the body becomes stronger and more capable by the next workout. If recovery is too short, the next workout consumes even more of the body’s resources, leading to overtraining. If recovery is too long, the supercompensation window closes, and progress is lost.
This is the foundational concept of periodisation: cycle training, stress, and recovery to maximise adaptation.
Higher HRV indicates more parasympathetic nervous system activity, less stress, and better recovery. Lower HRV indicates more sympathetic activation, more stress, and incomplete recovery. Decreased HRV has been shown to predict mortality after myocardial infarction, cancer, and sudden cardiac death, and is associated with heart failure, diabetic neuropathy, and liver cirrhosis. The risk of dying after a heart attack is more than 5-fold higher for those with low HRV vs. high HRV.
If your HRV is low, it isn’t a good idea to train hard or impose other hormetic stressors (sauna, cold exposure), since there’s an increase in sympathetic drive during and potentially after the acute stressful event. Sauna sessions may lower HRV acutely but have been shown to reduce arrhythmias and improve HRV in patients with chronic heart failure. Sauna and cold exposure can have cardiovascular benefits long-term, while looking neutral or negative on HRV in the short-term. Context matters; one HRV reading isn’t a diagnosis.
There’s a clear association between elevated pro-inflammatory IL-6, higher C-reactive protein, and decreased HRV. Elevated heart rate and body temperature, combined with low HRV, are often a sign of an active infection.
Things that lower HRV and impair recovery:
Things that raise HRV:
Varying training volume and intensity to achieve optimal performance while avoiding overtraining.
Train with weights light enough for proper form. Add 2.5kg per session for squat and deadlift; for other exercises, add weight every other session. Continue adding weight until you can no longer complete 3 × 5. Reduce set weights to what they were 2-3 weeks ago and begin again from there (a deload-and-reset).
The deep mechanistic breakdown of overtraining hypotheses (glycogen depletion, CNS fatigue, glutamine, oxidative stress, ANS imbalance, hypothalamus changes, cytokine storm) lives in The Exercise Rabbit Hole for those interested in the underlying physiology.
Pre-workout (if you train hard enough to warrant it):
Post-workout:
Antioxidant supplements, NSAIDs, and cryotherapy after working out reduce inflammation and exercise-induced oxidative stress, which slows or completely negates the adaptive signal needed for growth and improvement. Pre-workout antioxidant supplementation has been shown to interfere with mitochondrial biogenesis (a key endurance adaptation). Multiple studies have found that antioxidant supplementation impairs anabolic signalling and muscle hypertrophy. The popular framing that “more antioxidants are better” is wrong for trained athletes; some oxidative stress from training is the signal that drives adaptation. Save the antioxidants for periods of high stress or illness; reduce or skip them around training sessions.
Caveat: High-quality food will always beat supplementation, so don’t obsess over them. Seriously… The desire for convenience and shortcuts has made us nuts.
A brief note before we get into the material: the full write up on fasting and metabolic flexibility lives in Fasting in Part II. This section covers specifically the training-while-fasted dimension. When the Fasting page is rebuilt, some of this material will move there and be cross-referenced; for now, it lives here.
To lose fat, you have to be in an energy deficit (burn more than you consume). To build muscle, the conventional framing is that you need to be in an energy surplus (consume more than you burn).
Calories aren’t just calories because they can be partitioned differently depending on macronutrient ratios, nutrient quality, hormone levels, training status, and overall energetic demands on the body. If your workouts stimulate the muscles sufficiently, and you follow with adequate muscle protein synthesis by consuming enough protein, the rest of what you need for growth can come from stored body fat. You can also gain fat and lose muscle simultaneously by doing chronic cardio for hours and overconsuming calories with very little protein.
The adipose tissue consists of stored triglycerides (three fatty acid chains and a glycerol backbone). This single fat particle can cover most of the body’s metabolic needs in the short term. Fat is fuel that most tissues and muscles can use.
Lactate is the byproduct of glucose metabolism and contributes up to 18% of skeletal muscle glycogen synthesis after high-intensity exercise. During high-intensity workouts, you produce lactic acid by burning muscle glycogen. To eliminate the burn effect and restore lost glycogen, the body uses some of that lactate for muscle glycogen resynthesis.
There are times the body needs more fuel and amino acids than others:
In the case of intermittent fasting, you would see a much bigger lean muscle gain if you consumed most of your calories after a resistance training workout. The dominos are all set up: mechanical overload from exercise, depleted glycogen stores, activated mTOR, nervous system fatigue, and the conditions are favourable for building muscle as long as you stimulate muscle protein synthesis and bring in the building blocks.
Intermittent fasting and time-restricted feeding can be effective tools for building muscle and burning fat simultaneously:
Growth hormone increases substantially during fasting; older research suggested 2000-3000% increases at the 24-hour mark, though more recent work has shown the relative increase is more variable than this older framing suggests. Testosterone is another anabolic hormone affected favourably by short-term fasting:
Why might you lose muscle while fasting?
A ketogenic diet preserves muscle through several mechanisms:
Can fasting build muscle?
Studies have found fasting lowers the expression of mTOR and IGF-1, both needed for cellular growth, by increasing one of their inhibiting proteins (IGFBP1). mTOR is anabolic but also anti-catabolic. It protects the body against the harmful effects of cortisol and glucocorticoids on muscle tissue.
The rationale for trying to build muscle with intermittent fasting isn’t to maximise muscle growth or performance. It’s to prioritise longevity and avoid over-stimulating the anabolic effects of mTOR all the time.
When resistance training, you induce damage to muscle cells and tissues. If you consume adequate protein after the workout, you heal that damage and produce sarcoplasmic and myofibrillar hypertrophy. However, when working out fasted, you have limited amino acid availability and are subject to increased muscle damage. This may not be ideal for someone trying to build maximum muscle because they may end up with a NET negative muscle homeostasis.
The “anabolic window” panic from older bodybuilding literature has been substantially retracted; current research suggests post-workout protein intake within 2-3 hours is sufficient for maximal muscle protein synthesis, and that 24-hour total protein intake is the variable that counts more than acute timing. That said, some amino acids in circulation during a workout may minimise muscle damage and promote MPS afterwards.
Advantages of training fasted:
To maximise the autophagic benefits of fasting, fast for as long as possible each day. In most cases, that means about a 20-hour fasted window.
For optimal muscle growth, have a small amount of amino acids in your system before working out.
For better body composition and nutrient partitioning, backload most of your calories into the post-workout window where your body prioritises recovery.
Here’s what the protocol looks like:
After several thousand words of methodology, the practical takeaways come down to a manageable list:
For most adults pursuing general fitness:
Testing baseline:
Progressing over months and years:
Where to dig deeper:
The training methodology is rich, and the protocols are well-established. The hard part isn’t knowing what to do; it’s doing it consistently for years and decades. The framework’s general position on training is the same as its position on nutrition and sleep: foundational principles applied with consistency over time beat elaborate protocols applied inconsistently. Build the aerobic base. Lift heavy things twice a week. Move every day. Sleep well. Eat well. Re-test occasionally. Adjust based on what you find. Don’t fight your biology.